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Letter to the Editor

Common IL-31 Gene Haplotype Associated with Non-atopic Eczema is Not Implicated in Epidermolysis Bullosa Pruriginosa

doi: 10.2340/00015555-0958

Abstract:

Abstract is missing (Letter)

Authors:

Nikoletta Nagy, Akio Tanaka, Tanasit Techanukul, John A. McGrath

References

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  • Fine JD, Eady RA, Bauer JW, Bruckner-Tuderman L, Heagerty A, Hintner H, et al. The classification of inherited epidermolysis bullosa: report of the third international consensus meeting on the diagnosis and classification of epidermolysis bullosa. J Am Acad Dermatol 2008; 58: 931–950.
  • Mellerio JE, Ashton GH, Mohammedi R, Lyon CC, Kirby B, Harman KE, et al. Allelic heterogeneity of dominant and recessive COL7A1 mutations underlying epidermolysis bullosa pruriginosa. J Invest Dermatol 1999; 112: 984–987.
  • Almaani N, Liu L, Harrison N, Tanaka A, Lai-Cheong J, Mellerio JE, et al. New glycine substitution mutations in type VII collagen underlying epidermolysis bullosa pruriginosa but the phenotype is not explained by a common polymorphism in the matrix metalloproteinase-1 gene promoter. Acta Derm Venereol 2009; 89: 6–11.
  • Dang N, Murrell DF. Mutation analysis and characterization of COL7A1 mutations in dystrophic epidermolysis bullosa. Exp Dermatol 2008; 17: 553–568.
  • Schumann H, Has C, Kohlhase J, Bruckner-Tuderman L. Dystrophic epidermolysis bullosa pruriginosa is not associated with frequent FLG gene mutations. Br J Dermatol 2008; 159: 464–469.
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  • Dillon SR, Sprecher C, Hammond A, Bilsborough J, Rosenfeld-Franklin M, Presnell SR, et al. Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice. Nat Immunol 2004; 5: 752–760.
  • Raap U, Wichmann K, Bruder M, Ständer S, Wedi B, Kapp A, et al. Correlation of IL-31 serum levels with severity of atopic dermatitis. J Allergy Clin Immunol 2008; 122: 421–423.
  • Neis MM, Peters B, Dreuw A, Wenzel J, Bieber T, Mauch C, et al. Enhanced expression levels of IL-31 correlate with IL-4 and IL-13 in atopic and allergic contact dermatitis. J Allergy Clin Immunol 2006; 118: 930–937.
  • Sonkoly E, Muller A, Lauerma AI, Pivarcsi A, Soto H, Kemeny L, et al. IL-31: a new link between T cells and pruritus in atopic skin inflammation. J Allergy Clin Immunol 2006; 117: 411–417.
  • Zhang Q, Putheti P, Zhou Q, Liu Q, Gao W. Structures and biological functions of IL-31 and IL-31 receptors. Cytokine Growth Factor Rev 2008; 19: 347–356.
  • Tanaka A, Lai-Cheong JE, van den Akker PC, Nagy N, Millington G, Diercks GF, et al. The molecular skin pathology of familial primary localized cutaneous amyloidosis. Exp Dermatol 2010; 19: 416–423.
  • Schulz F, Marenholz I, Fölster-Holst R, Chen C, Sternjak A, Baumgrass R, et al. A common haplotype of the IL-31 gene influencing gene expression is associated with nonatopic eczema. J Allergy Clin Immunol 2007; 120: 1097–1102.
  • Grimstad O, Sawanobori Y, Vestergaard C, Bilsborough J, Olsen UB, Grønhøj-Larsen C, et al. Anti-interleukin-31-antibodies ameliorate scratching behaviour in NC/NgA mice: a model of atopic dermatitis. Exp Dermatol 2009; 18: 35–43.
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    Volume 90, Issue 6

    DOI: 10.2340/00015555-0958

    Pages: 631-632

    View at PubMed